We think of the Pineal
Gland, and its major hormone, Melatonin, as the primary coordinator of our
sleep-wake cycle. Melatonin is also
a cardiac-friendly hormone, with antioxidant, anti-hypertensive, and
anti-platelet effects (MI risk is lowest at 2 AM and highest at 9 AM).
A role of the Pineal gland and Melatonin in aging and senescence is also
appreciated. As we age, we make
less Melatonin; our body slows down, organ system dysfunction develops, and we
make less Melatonin, and so on. A
role for Melatonin in the regulation of energy production and tissue
calcification has been proposed: When
Melatonin is plentiful, it is easy to incorporate phosphorus into ATP, and
cellular energy is plentiful. When
Melatonin is in short supply, ATP production is blunted.
The phosphorus will then be incorporated into calcium pyrophosphate, and
the mitochondria will begin to calcify. This
process occurs first in energy intensive organs, such as the heart, the kidney,
and the pineal gland itself. As the
pineal begins to calcify, its output of Melatonin falls off further, so pineal
and extra-pineal calcification will progress.
The Pineal gland appears to be the first organ to calcify in man – the
beginning of the end. The Pineal
gland dysfunction → Melatonin deficiency → impaired energy
production → cellular calcification hypothesis follows the correct time
line, but it leaves out the all-important first step.
the knowledge that N. sanguineum is present in kidney stones, PKD cyst fluid and
tissue, and dental pulp stones, Hjelle looked for the presence of Nanobacterial
antigen in human pineal calcifications, called acervuli or “brain sand”.
Following EDTA pre-treatment, all acervuli specimens reacted (100% positive)
with NB 8/0 monoclonal antibody to N. sanguineum (the same monoclonal antibody
used in the PKD study, developed by Kajander and Ciftcioglu). N. sanguineum fixes calcium and phosphorus, producing
pathological calcification in the tissues affected, and N. sanguineum is clearly
present in the Pineal Gland. When
Nanobacteria are injected into rabbits, they can be recovered from the urine
within 15 minutes, and from the Cerebrospinal Fluid at one year.
Nanobacteremia and Nanobacteriuria are common in humans; the bugs cross
the placenta and blood-brain barrier and contaminate vaccines and biological
products made in fetal bovine serum. Could it be that N. sanguineum puts a brake
on human longevity, or that impaired immune function or recurrent Nanobacteremia
leads to organ system dysfunction and premature senescence, via the mechanism of
Pineal Gland calcification? Could
they be everywhere? Rather, why wouldn’t we assume that they are everywhere in
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